The endogenous psychoactive idea is fascinating. My friend called me during a psychosis, and I was convinced he was having a really bad psilocybin trip.
Modern research has actually found closer ties between psychosis and a different psychoactive compound: dynorphin[1]. In terms of recreational drugs, this is the neurotransmitter responsible for a salvia trip.
From an outside standpoint, this makes more sense than the classical psychedelics. Unlike LSD or psilocybin, salvia hallucinations usually result in a total disconnect from reality, and the user is often rendered catatonic like in severe cases of schizophrenia.
I remember some of the first time I did salvia. I was sitting on a couch at the end of a room, and I looked up at the corner of the ceiling, where the ceiling met two walls. Then I looked back at the people I was sitting with, but all I could see was the corner of the ceiling, where the ceiling met two walls. Then after... seconds? A minute? An hour? I could see normally again, but the room I was in began to move backward. It detached from an enormous wall, where it was revealed to be one of an infinite grid of identical rooms.
I don't remember the rest. Apparently all I did the whole time was stare with a blank expression for five minutes.
>Unlike LSD or psilocybin salvia hallucinations salvia . . . user is often rendered catatonic.
Woah! Granted I’ve see only seen a handful of people use it, but it’s the only drug which I seriously worry about it causing agitation/frenzy so severe as to induce outward violence.
